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Day · Friday 4 December 2026

Contraction

Contraction

Contraction

Diabetes as a model problem

Diabetes as a model problem

Second day of the seminar. It takes type 2 diabetes as a model problem: its reading as a coordination failure across scales, insulin resistance, windows of reversal and therapeutic targets.

Benefits

Read type 2 diabetes as a problem of coordination across scales

Locate insulin resistance in the component of the system that sustains it

Recognise the windows in which a metabolic condition admits reversal

Identify the therapeutic targets the current arsenal leaves open

Apply the same reading to other chronic conditions

Session

Multiscale coordination failure

Multiscale coordination failure

Type 2 diabetes as a model problem

In type 2 diabetes the healthy state can be reached under forced intervention, and counter-regulation shows that the effectors respond: the machinery is there. The session takes the best-documented metabolic condition, reads it component by component and locates the failure in coordination rather than in capacity.

Failure profile by component: states and operators preserved; constraints, evaluation and horizon degraded

Evidence that the machinery is preserved: a reachable healthy state and active counter-regulation

Nine independent mechanisms that converge on coordination rather than on quantity

The three targets: intercellular coupling in the islet, hepato-vagal afference and the hypothalamic reference circuit

Session

Insulin resistance

Insulin resistance

Molecular mechanisms and therapeutic consequences

About half of postprandial glucose uptake depends on a hepatic signal that is released only under parasympathetic permission: that neural signal releases nothing on its own, but without it the chemical signal has no effect. The session develops that permission and the different mechanisms that can lie beneath one and the same clinical finding.

Hepatic parasympathetic innervation: the permission that enables insulin-mediated glucose disposal without producing an effect of its own

Dependence on the prandial state: the same insulin dose releases the signal after a meal and not while fasting

Dependence on the route of entry: action abolished by hepatic denervation and restored by intraportal, not intravenous, infusion

Closure of the permission while fasting as a protective constraint against hypoglycaemia

Mechanisms distinguishable by the observed response: reduced per unit of signal, absent with sufficient signal, or present only in one context

A permission that stops activating after meals: a correct constraint in the wrong context, and its reversibility

Session

Reversibility in type 2 diabetes

Reversibility in type 2 diabetes

Ratchets and windows of reversal

The state a system reaches rewrites what it is still permitted to do, and some of those rewritings are not undone when the state returns. The session orders the mechanisms documented in type 2 diabetes by whether they admit reversal and over what time frame.

Arrows from state to constraint: how the state reached changes what the system is permitted to do

Ratchet: what distinguishes a rewriting that can be undone from one that cannot

Ordering the mechanisms by reversibility and time window

Windows of reversal: from days to weeks, and those that depend on restoring a physiological cycle

The cost of merging mechanisms with different reversibility: recoverable targets given up as lost

Session

Therapeutic strategies

Therapeutic strategies

What the current arsenal does not address and which targets remain

Type 2 diabetes has one of the most complete arsenals in internal medicine and remains progressive. The session examines what is left unaddressed when treatment is ordered by the component it repairs, and which strategies follow from that reading.

What routine monitoring measures: the glucose level reached, not the capacity to regulate it

Continuous exposure versus pulsatile exposure anchored to meals

Targets by component: anticipatory response, intercellular coupling in the islet and microbiota

Combination criterion: covering different points of the failure rather than adding effect on the same one

Regulation programmes built from the quintuple: pharmacological and non-pharmacological interventions ordered by the component they repair

Carrying the reading over to other chronic conditions: dyslipidaemia, metabolic syndrome and the thyroid axis

Venue

Hotel Galería Plaza Reforma, Mexico City

Hours

10:00 to 18:00

Format

In person and online, with live streaming

Fees

Available via WhatsApp

Enquiries

Request information on WhatsApp

+52 55 7907 6626 · contacto@institutocentrobioenergetica.com

Instituto Centrobioenergetica · Mexico City

Diverse Cognition 2026

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© 2026 Instituto Centrobioenergética

© 2026 Instituto Centrobioenergética

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