El nodo de lesión

Selección de la evidencia fisiológica y biofísica que fundamenta el modelo.

1. Nilsson P, et al. Regional changes in interstitial K⁺ and Ca²⁺ following cortical contusion trauma. J Cereb Blood Flow Metab. 1993;13(2):183-92.
2. Katayama Y, et al. Massive increases in extracellular potassium after concussive brain injury. J Neurosurg. 1990;73(6):889-900.
3. Peracchia C. Calmodulin-mediated regulation of gap junction channels. Int J Mol Sci. 2020;21(2):485.
4. McLean MJ, et al. Blockade of sensory neuron action potentials by a static magnetic field in the 10 mT range. Bioelectromagnetics. 1995;16(1):20-32.
5. Cavopol AV, et al. Static magnetic fields that block action potentials in cultured neurons. Bioelectromagnetics. 1995;16(3):197-206.
6. Morris C, Skalak T. Static magnetic fields alter arteriolar tone in vivo. Bioelectromagnetics. 2005;26(1):1-9.
7. Rosen AD. Mechanism of action of moderate-intensity static magnetic fields on biological systems. Cell Biochem Biophys. 2003;39(2):163-173.
8. Fan Y, et al. The analgesic effects of static magnetic fields. Bioelectromagnetics. 2021;42(2):115-127.
9. Rosas-Ballina M, et al. Acetylcholine-synthesizing T cells relay neural signals in a vagus nerve circuit. Science. 2011;334(6052):98-101.
10. Palmer BF. Regulation of potassium homeostasis. Clin J Am Soc Nephrol. 2014;10(6):1050-1060.
11. Milano S, et al. β3-adrenoceptor in the sympathetic regulation of renal acid-base homeostasis. Front Physiol. 2024;15:1304375.
12. Levin M. Bioelectric signaling: reprogrammable circuits underlying embryogenesis, regeneration and cancer. Cell. 2021;184(8):1971-1989.